Paradoxical Reaction in a Patient with Hidradenitis Suppurativa Undergoing Adalimumab Treatment
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https://doi.org/10.2340/00015555-3844Keywords:
inflammation, interleukin-17, myxovirus resistance protein 1Abstract
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Conrad C, Domizio JD, Mylonas A, Belkhodja C, Demaria O, Navarini AA, et al. TNF blockade induces a dysregulated type I interferon response without autoimmunity in paradoxical psoriasis. Nat Commun 2018; 9: 25.
DOI: https://doi.org/10.1038/s41467-017-02466-4
Collamer AN, Guerrero KT, Henning JS, Battafarano DF. Psoriatic skin lesions induced by tumor necrosis factor antagonist therapy: a literature review and potential mechanisms of action. Arthritis Rheum 2008; 59: 996-1001.
DOI: https://doi.org/10.1002/art.23835
Mylonas A, Conrad C. Psoriasis: classical vs. paradoxical. The yin-yang of TNF and type I interferon. Front Immunol 2018; 9: 2746.
DOI: https://doi.org/10.3389/fimmu.2018.02746
Morelli M, Scarponi C, Madonna S, Albanesi C. Experimental methods for the immunological characterization of paradoxical psoriasis reactions induced by TNF-? biologics. Methods Mol Biol 2021; 2248: 155-165.
DOI: https://doi.org/10.1007/978-1-0716-1130-2_11
Dequidt L, Cogrel O, Guillet S, Milpied B, Darrigade A S, Beylot-Barry M, et al. Paradoxical psoriasiform reactions to antitumour necrosis factor-alpha drugs in hidradenitis suppurativa. Br J Dermatol 2018; 178: 281-283.
DOI: https://doi.org/10.1111/bjd.15616
Vural S, Gündo?du M, Gökp?nar IE, Durmaz CD, Vural A, Steinmüller-Magin L, et al. Association of pyrin mutations and autoinflammation with complex phenotype hidradenitis suppurativa: a case-control study. Br J Dermatol 2019; 180: 1459-1467.
DOI: https://doi.org/10.1111/bjd.17466
Nakazawa Y, Sasaki K, Mitsutake N, Matsuse M, Shimada M, Nardo T, et al. Mutations in UVSSA cause UV-sensitive syndrome and impair RNA polymerase IIo processing in transcription-coupled nucleotide-excision repair. Nat Genet 2012; 44: 586-592.
DOI: https://doi.org/10.1038/ng.2229
Nomura T. Hidradenitis suppurativa as a potential subtype of autoinflammatory keratinization disease. Front Immunol 2020; 11: 847.
DOI: https://doi.org/10.3389/fimmu.2020.00847
Marzano AV, Damiani G, Ceccherini I, Berti E, Gattorno M, Cugno M. Autoinflammation in pyoderma gangrenosum and its syndromic form (pyoderma gangrenosum, acne and suppurative hidradenitis). Br J Dermatol 2017; 176: 1588-1598.
DOI: https://doi.org/10.1111/bjd.15226
Shoham NG, Centola M, Mansfield E, Hull KM, Wood G, Wise CA, et al. Pyrin binds the PSTPIP1/CD2BP1 protein, defining familial Mediterranean fever and PAPA syndrome as disorders in the same pathway. Proc Natl Acad Sci USA 2003; 100: 13501-13506.
DOI: https://doi.org/10.1073/pnas.2135380100
Nishimori N, Hayama K, Kimura K, Fujita H, Fujiwara K, Terui T. A novel NCSTN gene mutation in a Japanese family with hidradenitis suppurativa. Acta Derm Venereol 2020; 100: adv00283.
DOI: https://doi.org/10.2340/00015555-3632
Kelly G, Hughes R, McGarry T, van den Born M, Adamzik K, Fitzgerald R, et al. Dysregulated cytokine expression in lesional and nonlesional skin in hidradenitis suppurativa. Br J Dermatol 2015; 173: 1431-1439.
DOI: https://doi.org/10.1111/bjd.14075
Fania L, Morelli M, Scarponi C, Mercurio L, Scopelliti F, Cattani C, et al. Paradoxical psoriasis induced by TNF-alpha blockade shows immunological features typical of the early phase of psoriasis development. J Pathol Clin Res 2020; 6: 55-68.
DOI: https://doi.org/10.1002/cjp2.147
de Gannes GC, Ghoreishi M, Pope J, Russell A, Bell D, Adams S, et al. Psoriasis and pustular dermatitis triggered by TNF-? Inhibitors in patients with rheumatologic conditions. Arch Dermatol 2007;143; 2: 223-231.
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Copyright (c) 2021 Soichiro Ikeya, Takuya Takeichi, Tomoki Taki, Yoshinao Muro, Tomoo Ogi, Masashi Akiyama
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